Αυτά φυσικά γίνονται αφότου το 87% των Σκωτσέζων έχει εμβολιασθεί, κι οι εβδομαδιαίοι θάνατοι είναι κατά 30% πάνω από το αναμενόμενο για την εποχή. Μόνο το 1891 ήσαν τόσοι λόγω της Ρώσικης γρίπης.
Αντίστροφη μέτρηση για τη δήθεν ΑΝΟΣΙΑ η οποία αποδεικνύεται ΜΗΔΕΝΙΚΗ… τα θύματα εμβολίων θα βλέπουν την ανοσολογική τους αντίδραση να μειώνεται κατά περίπου 5% κάθε εβδομάδα, με μακροπρόθεσμες εκδηλώσεις να ομοιάζουν με τη νόσο του AIDS
Μια έκθεση βόμβα που δημοσιεύτηκε από το The Expose (Ηνωμένο Βασίλειο) έχει τον τίτλο: “Μια σύγκριση των επίσημων κυβερνητικών εκθέσεων υποδηλώνει ότι οι Πλήρως Εμβολιασμένοι αναπτύσσουν Σύνδρομο Επίκτητης Ανοσοανεπάρκειας.” Δεδομένου ότι αυτό υποκινείται από τις ενέσεις εμβολίου, το ονομάζω “Σύνδρομο ενέσιμης ανοσοανεπάρκειας” ή “IIDS Injection, ImDeficiency Syndrome “.
Όπως εξηγεί το The Expose, μετά από μια λεπτομερή ανάλυση των δεδομένων παρακολούθησης εμβολίων PHE του Ηνωμένου Βασιλείου:
Τα τελευταία στοιχεία της βρετανικής έκθεσης PHE Vaccine Surveillance Report για τα κρούσματα Covid δείχνουν ότι τα διπλά εμβολιασμένα άτομα 40-70 ετών έχουν χάσει το 40% της ικανότητάς τους στο ανοσοποιητικό σύστημα σε σύγκριση με τους μη εμβολιασμένους. Το ανοσοποιητικό τους σύστημα επιδεινώνεται περίπου στο 5% την εβδομάδα (μεταξύ 2,7% και 8,7%). Εάν συνεχιστεί αυτό, τότε τα παιδιά και οι ηλικίες 30-50 ετών θα έχουν 100% υποβάθμιση του ανοσοποιητικού τους συστήματος, μηδενική ιική άμυνα μέχρι τα Χριστούγεννα και όλοι οι διπλά εμβολιασμένοι άνω των 30 ετών θα έχουν χάσει το ανοσοποιητικό τους σύστημα μέχρι τον Μάρτιο του επόμενου έτους.
Σύμφωνα με την τάση που περιγράφεται από τα τρέχοντα δεδομένα, όπως προσθέτει το The Expose:
Όλοι άνω των 30 ετών θα έχουν χάσει το 100% ολόκληρης της ανοσολογικής τους ικανότητας (για ιούς και ορισμένους καρκίνους) μέσα σε 6 μήνες. Τα παιδιά και οι ηλικίες 30-50 ετών θα το έχουν χάσει μέχρι τα Χριστούγεννα. Αυτοί οι άνθρωποι τότε θα έχουν αποκτήσει ουσιαστικά πλήρες σύνδρομο επίκτητης ανοσοανεπάρκειας και θα καταστρέψουν το NHS (το εθνικό Σύστημα Υγείας της Αγγλίας).
Οι ενισχυτικές λήψεις επιταχύνουν περαιτέρω την καταστροφή του ανοσοποιητικού συστήματος, γεγονός που εξηγεί γιατί το 90% των εισαγωγών σε νοσοκομεία σε τουλάχιστον ένα νοσοκομείο των ΗΠΑ ήταν μεταξύ εμβολιασμένων ατόμων, σύμφωνα με έναν νέο πληροφοριοδότη που προχώρησε μέσω του The Highwire . Με πίστωση στο TheHighWire.com , έχουμε δημοσιεύσει αυτό το βίντεο πληροφοριοδοτών εδώ:
Εάν η τάση διατηρηθεί, τους επόμενους έξι μήνες θα δούμε ένα παγκόσμιο κύμα θυμάτων εμβολίων να πεθαίνουν από καρκίνο και κοινές λοιμώξεις
Η κατακόρυφη πτώση στην ανοσολογική απάντηση μεταξύ των θυμάτων του εμβολίου σημαίνει ότι οι θάνατοι από καρκίνο – και από κοινές λοιμώξεις όπως τα χειμερινά κρυολογήματα και η γρίπη – πρόκειται να εκτοξευθούν στα ύψη τους επόμενους έξι μήνες. Όλοι έχουμε ένα πλήρως λειτουργικό ανοσοποιητικό σύστημα, άλλωστε, που διατηρεί τον καρκίνο υπό έλεγχο και αποτρέπει τα κοινά παθογόνα άγριου λοιμωγόνου τύπου να κάνουν καταστροφές σε όλο το σώμα.
Η ανοσοποιητική λειτουργία υποστηρίζεται και ενισχύεται από τη βιταμίνη D, τον ψευδάργυρο και άλλα κρίσιμα θρεπτικά συστατικά, γι ‘αυτό ακριβώς τα γενοκτονικά του πληθυσμού Μέσα Μαζικής Επικοινωνίας και τα λεγόμενα “επιστημονικά” περιοδικά επιτίθενται στη διατροφή (καθώς και στη χρήση ιβερμεκτίνης-Scaball). Μια ανεπάρκεια θρεπτικών συστατικών επηρεάζει τη λειτουργία του ανοσοποιητικού, γι ‘αυτό τα άτομα με ανεπάρκεια βιταμίνης D τείνουν να αρρωσταίνουν πολύ πιο συχνά από τα άλλα. Και τώρα, με τα εμβόλια να καταστρέφουν κυριολεκτικά τη λειτουργία του ανοσοποιητικού από εβδομάδα σε εβδομάδα, έχουμε τη σκόπιμη κατάργηση της ανθρώπινης ανοσολογικής λειτουργίας σε συνδυασμό με διατροφικές ελλείψεις και τεχνητούς περιορισμούς στις ιατρικές παρεμβάσεις που μπορεί να σώσουν ζωές.
Η μόνη αληθοφανής εξήγηση για όλα αυτά είναι ότι τα εμβόλια είναι, πράγματι, ένα σύστημα απελευθέρωσης όπλων ενάντια στον πληθυσμό και λειτουργούν καταστρέφοντας το ανοσοποιητικό σύστημα ενός θύματος, προκαλώντας το θάνατό του από καρκίνο ή κοινές λοιμώξεις. Μόλις σήμερα, ο πλήρως εμβολιασμένος Κόλιν Πάουελ πέθανε από τον κορωνοϊό, προσθέτοντας ένα ακόμη θύμα από το εμβόλιο στην ατζέντα του αποπληθυσμού.
Όπως γνωρίζουν πολύ καλά οι ασθενείς με AIDS, εκείνα τα άτομα που πάσχουν από βαριά κατεσταλμένη ανοσολογική λειτουργία μπορεί να σκοτωθούν από συνηθισμένα χειμερινά κρυολογήματα ή απλούς παθογόνους παράγοντες όπως της γρίπης που κανονικά δεν θα δημιουργούσαν κανένα απολύτως κίνδυνο για ένα υγιές άτομο. Δεν γνωρίζαμε ότι ένα οπλικό σύστημα παρόμοιο με το AIDS θα φορτωνόταν σε βελόνες και ανήθικα θα ονομαζόταν «εμβόλια και εμβολιασμοί» για να ξεγελάσει τους αδύναμους και ευκολόπιστους.
Η κακή ιδιοφυΐα σε αυτό το παγκόσμιο σχέδιο αποπληθυσμού είναι ότι οι θάνατοι που προκαλούνται από το εμβόλιο ένεκα τηςπροκαλούμενης από αυτά ανοσολογικής καταστροφής δεν θα αποδοθούν ποτέ στο ίδιο το εμβόλιο . Όσοι πεθαίνουν από καρκίνο αφού τα εμβόλια κατέστρεψαν το ανοσοποιητικό τους σύστημα θα καταγραφούν ως θάνατοι από «καρκίνο» και όχι θάνατοι από εμβόλια. Και όσοι πεθαίνουν από κοινές λοιμώξεις ή στελέχη γρίπης θα περιγραφούν ότι έχουν μολυνθεί από στελέχη ενός κύματος «υπεργρίπης», για τους οποίους, φυσικά, θα κατηγορηθούν ότι φταίνε τα μη εμβολιασμένα άτομα.
Ακόμα κι αν εκατομμύρια Αμερικανοί πεθάνουν τους επόμενους έξι μήνες και τα νοσοκομεία υπερβούν εντελώς τιθς δυνατότητες εισαγωγής ασθενών, τα εμβόλια για τον κορονοϊό δεν θα συνδεθούν ποτέ με κανένα εμβόλιο. Τα ΜΜΕ που χρηματοδοτούνται από τα φαρμακευτικά μέσα θα το βεβαιώνουν ακατάπαυστα κι επαναλαμβανόμενα.
Αναμένεται ότι τα νοσοκομεία των ΗΠΑ θα βρίσκονται σε κατάσταση έκτακτης ανάγκης και θα ανεπαρκούν να καλύψουν τους εισαγόμενους έστω και με πλήρη ανάπτυξη μέχρι τις αρχές του 2022.
Ακόμα χειρότερα, όταν οι νοσηλευτές και οι εργαζόμενοι στα νοσοκομεία με φυσική ανοσία απολύονται αυτή τη στιγμή από τη δουλειά τους επειδή αρνούνται να κάνουν ενέσεις spike πρωτεΐνης της ακίδας . Αυτό προκαλεί σοβαρές ελλείψεις προσωπικού σε ορισμένες περιοχές (όπως η Νέα Υόρκη) και δεν έχουμε ακόμη μπει στην εποχή της γρίπης. Μόλις τα επίπεδα της βιταμίνης D πέσουν κατά τους σκοτεινότερους, κρύους μήνες Δεκέμβριο, Ιανουάριο και Φεβρουάριο, είναι πολύ πιθανό να δούμε τα νοσοκομεία σε πανικό προσωπικού την ίδια στιγμή που ξεκινά το κύμα των θυμάτων εμβολίων που έχουν υποστεί βλάβη στο ανοσοποιητικό σύστημα.
Προσέξτε τα ψέματα των εταιρειών που αποκαλούνται Μέσα Μαζικής Ενημέρωσης να αποκαλύπτουν πολλές ιστορίες της κρίσης στα νοσοκομεία, υποστηρίζοντας ότι «οι άνθρωποι πεθαίνουν επειδή οι εγωιστές αντι-εμβολιαστές δεν θα κάνουν τα εμβόλιά τους!» Στην πραγματικότητα, όμως, θα είναι οι εμβολιασμένοι που θα πεθάνουν από τις δικές τους σφαίρες αυτοκτονίας , καθώς το να επιτρέπετε στον εαυτό σας να εγχέεται ένα θανατηφόρο βιολογικό όπλο με νανοσωματίδια δια της πρωτεΐνης αιχμής δεν είναι η καλλίτερη και η πιο σοφή στρατηγική για να διατηρήσετε την υγεία σας.
Το αποτέλεσμα είναι η πανδημία του covid είναι ουσιαστικά και ρεαλιστικά ένα παγκόσμιο σχέδιο αποπληθυσμού και η υστερία σχετικά με τον covid σχεδιάστηκε για να ωθήσει τους ανθρώπους να δεχτούν αυτά τα θανατηφόρα εμβόλια που καταστρέφουν την ανοσοποιητική τους λειτουργία και τους βυθίζουν σε σχεδόν βέβαιο θάνατο. Οι επόμενοι έξι μήνες θα αποκαλύψουν μαζικό θάνατο σε ολόκληρο το βόρειο ημισφαίριο, ακόμη και αν οι εγκληματίες πολέμου προωθούν αυτά τα θανατηφόρα εμβόλια και προτείνουν να διπλασιαστούν λόγω των γενοκτονικών απαιτήσεών τους.
Ο λόγος που θέλουν το 100% του κοινού να εμβολιαστεί είναι ώστε με αυτό τον τρόπο να μην υπάρχει ομάδα ελέγχου που να μπορέσει να συγκριθεί με την ομάδα των εμβολιασμένων. Όσο υπάρχει μια ομάδα ελέγχου απαλλαγμένων ατόμων, η θνησιμότητα που σχετίζεται με τις ενέσεις εμβολίων δεν μπορεί να περάσει κάτω από το χαλί. Στην πραγματικότητα, όπως αναφέρει τώρα ο Alex Berenson, υπάρχουν υπερβολικοί θάνατοι μετά το εμβόλιο στη Σκωτία καθώς και σε ολόκληρη τη Γερμανία .
Το παρακάτω άρθρο έκανε εντύπωση ολοσχερώς αρνητική στην ιατρό , ερευνήτια και συγγραφέα Meryl Nass η οποία το βρήκε όχι απλώς ατελές αλλά ότι κάτι δεν πήγαινε και αναρωτήθηκε πως τόσου ευηπόληπτοι και καταξιωμένοι συνάδελφοι ήταν δυνατόν να χρησιμοποιήσουν αυτό το άρθρο για να δικαιολογήσουν και να δικαιολογούν μέχρι σήμερα κάτι που ήταν εξ αρχής καταφανές γι’ αυτήν…
Πως ο ιός κατασκευάστηκε πειραματικά σε εργαστήριο με συγκεκριμένους στόχους.
Και σαν επιστέγασμα ακολούθησε ο Νομπελίστας Luk Montagnier ο οποίος είπε: ¨Εγώ μιλώ με καθαρά επιστημονικά δεδομένα και όχι υποθέσεις. Η αλληλουχία του γονιδιώματος του Sars-COV-2 δείχνει ανθρώπινη επέμβαση. Πρόκειται για τεχνικά κατασκευασμένο ιό”. Δείτε το παρακάτω βίντεο αποκαλούμενο “ΠΑΝΔΗΜΙΑ” και αφού διαβάσετε το πιο κάτω δήθεν επιστημονικό άρθρο-δικαιολογία, βγάλτε μόνοι σας το συμπέρασμα για την παγκόσμια απάτη που λέγεται πανδημία εκ κορωνοϊού.
Το πολύ γνωστό περιοδικό Nature Medicine διαφωνεί, ο κορωνοϊός είναι αποτέλεσμα μεταλλάξεων από ζώο (νυχτερίδα) και μόλυνσης του ανθρώπου παρά τεχνητής τροποποίησης.
To the Editor — Since the first reports of novel pneumonia (COVID-19) in Wuhan, Hubei province, China1,2, there has been considerable discussion on the origin of the causative virus, SARS-CoV-23 (also referred to as HCoV-19)4. Infections with SARS-CoV-2 are now widespread, and as of 11 March 2020, 121,564 cases have been confirmed in more than 110 countries, with 4,373 deaths5.
SARS-CoV-2 is the seventh coronavirus known to infect humans; SARS-CoV, MERS-CoV and SARS-CoV-2 can cause severe disease, whereas HKU1, NL63, OC43 and 229E are associated with mild symptoms6. Here we review what can be deduced about the origin of SARS-CoV-2 from comparative analysis of genomic data. We offer a perspective on the notable features of the SARS-CoV-2 genome and discuss scenarios by which they could have arisen. Our analyses clearly show that SARS-CoV-2 is not a laboratory construct or a purposefully manipulated virus.
Notable features of the SARS-CoV-2 genome
Our comparison of alpha- and betacoronaviruses identifies two notable genomic features of SARS-CoV-2: (i) on the basis of structural studies7,8,9 and biochemical experiments1,9,10, SARS-CoV-2 appears to be optimized for binding to the human receptor ACE2; and (ii) the spike protein of SARS-CoV-2 has a functional polybasic (furin) cleavage site at the S1–S2 boundary through the insertion of 12 nucleotides8, which additionally led to the predicted acquisition of three O-linked glycans around the site.
1. Mutations in the receptor-binding domain of SARS-CoV-2
The receptor-binding domain (RBD) in the spike protein is the most variable part of the coronavirus genome1,2. Six RBD amino acids have been shown to be critical for binding to ACE2 receptors and for determining the host range of SARS-CoV-like viruses7. With coordinates based on SARS-CoV, they are Y442, L472, N479, D480, T487 and Y4911, which correspond to L455, F486, Q493, S494, N501 and Y505 in SARS-CoV-27. Five of these six residues differ between SARS-CoV-2 and SARS-CoV (Fig. 1a). On the basis of structural studies7,8,9 and biochemical experiments1,9,10, SARS-CoV-2 seems to have an RBD that binds with high affinity to ACE2 from humans, ferrets, (νυφίτσες), cats and other species with high receptor homology7.
Fig. 1: Features of the spike protein in human SARS-CoV-2 and related coronaviruses.
a, Mutations in contact residues of the SARS-CoV-2 spike protein. The spike protein of SARS-CoV-2 (red bar at top) was aligned against the most closely related SARS-CoV-like coronaviruses and SARS-CoV itself. Key residues in the spike protein that make contact to the ACE2 receptor are marked with blue boxes in both SARS-CoV-2 and related viruses, including SARS-CoV (Urbani strain). b, Acquisition of polybasic cleavage site and O-linked glycans. Both the polybasic cleavage site and the three adjacent predicted O-linked glycans are unique to SARS-CoV-2 and were not previously seen in lineage B betacoronaviruses. Sequences shown are from NCBI GenBank, accession codes MN908947, MN996532, AY278741, KY417146 and MK211376. The pangolin coronavirus sequences are a consensus generated from SRR10168377 and SRR10168378 (NCBI BioProject PRJNA573298)29,30.
While the analyses above suggest that SARS-CoV-2 may bind human ACE2 with high affinity, computational analyses predict that the interaction is not ideal7 and that the RBD sequence is different from those shown in SARS-CoV to be optimal for receptor binding7,11. Thus, the high-affinity binding of the SARS-CoV-2 spike protein to human ACE2 is most likely the result of natural selection on a human or human-like ACE2 that permits another optimal binding solution to arise. This is strong evidence that SARS-CoV-2 is not the product of purposeful manipulation.
2. Polybasic furin cleavage site and O-linked glycans
The second notable feature of SARS-CoV-2 is a polybasic cleavage site (RRAR) at the junction of S1 and S2, the two subunits of the spike8 (Fig. 1b). This allows effective cleavage by furin and other proteases and has a role in determining viral infectivity and host range12. In addition, a leading proline is also inserted at this site in SARS-CoV-2; thus, the inserted sequence is PRRA (Fig. 1b). The turn created by the proline is predicted to result in the addition of O-linked glycans to S673, T678 and S686, which flank the cleavage site and are unique to SARS-CoV-2 (Fig. 1b). Polybasic cleavage sites have not been observed in related ‘lineage B’ betacoronaviruses, although other human betacoronaviruses, including HKU1 (lineage A), have those sites and predicted O-linked glycans13. Given the level of genetic variation in the spike, it is likely that SARS-CoV-2-like viruses with partial or full polybasic cleavage sites will be discovered in other species.
The functional consequence of the polybasic cleavage site in SARS-CoV-2 is unknown, and it will be important to determine its impact on transmissibility and pathogenesis in animal models. Experiments with SARS-CoV have shown that insertion of a furin cleavage site at the S1–S2 junction enhances cell–cell fusion without affecting viral entry14. In addition, efficient cleavage of the MERS-CoV spike enables MERS-like coronaviruses from bats to infect human cells15. In avian influenza viruses, rapid replication and transmission in highly dense chicken populations selects for the acquisition of polybasic cleavage sites in the hemagglutinin (HA) protein16, which serves a function similar to that of the coronavirus spike protein. Acquisition of polybasic cleavage sites in HA, by insertion or recombination, converts low-pathogenicity avian influenza viruses into highly pathogenic forms16. The acquisition of polybasic cleavage sites by HA has also been observed after repeated passage in cell culture or through animals17.
The function of the predicted O-linked glycans is unclear, but they could create a ‘mucin-like domain’ that shields epitopes or key residues on the SARS-CoV-2 spike protein18. Several viruses utilize mucin-like domains as glycan shields involved immunoevasion18. Although prediction of O-linked glycosylation is robust, experimental studies are needed to determine if these sites are used in SARS-CoV-2.
Theories of SARS-CoV-2 origins
It is improbable that SARS-CoV-2 emerged through laboratory manipulation of a related SARS-CoV-like coronavirus. As noted above, the RBD of SARS-CoV-2 is optimized for binding to human ACE2 with an efficient solution different from those previously predicted7,11. Furthermore, if genetic manipulation had been performed, one of the several reverse-genetic systems available for betacoronaviruses would probably have been used19. However, the genetic data irrefutably show that SARS-CoV-2 is not derived from any previously used virus backbone20. Instead, we propose two scenarios that can plausibly explain the origin of SARS-CoV-2: (i) natural selection in an animal host before zoonotic transfer; and (ii) natural selection in humans following zoonotic transfer. We also discuss whether selection during passage could have given rise to SARS-CoV-2.
1. Natural selection in an animal host before zoonotic transfer
As many early cases of COVID-19 were linked to the Huanan market in Wuhan1,2, it is possible that an animal source was present at this location. Given the similarity of SARS-CoV-2 to bat SARS-CoV-like coronaviruses2, it is likely that bats serve as reservoir hosts for its progenitor. Although RaTG13, sampled from a Rhinolophus affinis bat1, is ~96% identical overall to SARS-CoV-2, its spike diverges in the RBD, which suggests that it may not bind efficiently to human ACE27 (Fig. 1a).
Malayan pangolins (Manis javanica) illegally imported into Guangdong province contain coronaviruses similar to SARS-CoV-221. Although the RaTG13 bat virus remains the closest to SARS-CoV-2 across the genome1, some pangolin coronaviruses exhibit strong similarity to SARS-CoV-2 in the RBD, including all six key RBD residues21 (Fig. 1). This clearly shows that the SARS-CoV-2 spike protein optimized for binding to human-like ACE2 is the result of natural selection.
Neither the bat betacoronaviruses nor the pangolin betacoronaviruses sampled thus far have polybasic cleavage sites. Although no animal coronavirus has been identified that is sufficiently similar to have served as the direct progenitor of SARS-CoV-2, the diversity of coronaviruses in bats and other species is massively undersampled. Mutations, insertions and deletions can occur near the S1–S2 junction of coronaviruses22, which shows that the polybasic cleavage site can arise by a natural evolutionary process. For a precursor virus to acquire both the polybasic cleavage site and mutations in the spike protein suitable for binding to human ACE2, an animal host would probably have to have a high population density (to allow natural selection to proceed efficiently) and an ACE2-encoding gene that is similar to the human ortholog.
2. Natural selection in humans following zoonotic transfer
It is possible that a progenitor of SARS-CoV-2 jumped into humans, acquiring the genomic features described above through adaptation during undetected human-to-human transmission. Once acquired, these adaptations would enable the pandemic to take off and produce a sufficiently large cluster of cases to trigger the surveillance system that detected it1,2.
All SARS-CoV-2 genomes sequenced so far have the genomic features described above and are thus derived from a common ancestor that had them too. The presence in pangolins of an RBD very similar to that of SARS-CoV-2 means that we can infer this was also probably in the virus that jumped to humans. This leaves the insertion of polybasic cleavage site to occur during human-to-human transmission.
Estimates of the timing of the most recent common ancestor of SARS-CoV-2 made with current sequence data point to emergence of the virus in late November 2019 to early December 201923, compatible with the earliest retrospectively confirmed cases24. Hence, this scenario presumes a period of unrecognized transmission in humans between the initial zoonotic event and the acquisition of the polybasic cleavage site. Sufficient opportunity could have arisen if there had been many prior zoonotic events that produced short chains of human-to-human transmission over an extended period. This is essentially the situation for MERS-CoV, for which all human cases are the result of repeated jumps of the virus from dromedary camels, producing single infections or short transmission chains that eventually resolve, with no adaptation to sustained transmission25.
Studies of banked human samples could provide information on whether such cryptic spread has occurred. Retrospective serological studies could also be informative, and a few such studies have been conducted showing low-level exposures to SARS-CoV-like coronaviruses in certain areas of China26. Critically, however, these studies could not have distinguished whether exposures were due to prior infections with SARS-CoV, SARS-CoV-2 or other SARS-CoV-like coronaviruses. Further serological studies should be conducted to determine the extent of prior human exposure to SARS-CoV-2.
3. Selection during passage
Basic research involving passage of bat SARS-CoV-like coronaviruses in cell culture and/or animal models has been ongoing for many years in biosafety level 2 laboratories across the world27, and there are documented instances of laboratory escapes of SARS-CoV28. We must therefore examine the possibility of an inadvertent laboratory release of SARS-CoV-2.
In theory, it is possible that SARS-CoV-2 acquired RBD mutations (Fig. 1a) during adaptation to passage in cell culture, as has been observed in studies of SARS-CoV11. The finding of SARS-CoV-like coronaviruses from pangolins with nearly identical RBDs, however, provides a much stronger and more parsimonious explanation of how SARS-CoV-2 acquired these via recombination or mutation19.
The acquisition of both the polybasic cleavage site and predicted O-linked glycans also argues against culture-based scenarios. New polybasic cleavage sites have been observed only after prolonged passage of low-pathogenicity avian influenza virus in vitro or in vivo17. Furthermore, a hypothetical generation of SARS-CoV-2 by cell culture or animal passage would have required prior isolation of a progenitor virus with very high genetic similarity, which has not been described. Subsequent generation of a polybasic cleavage site would have then required repeated passage in cell culture or animals with ACE2 receptors similar to those of humans, but such work has also not previously been described. Finally, the generation of the predicted O-linked glycans is also unlikely to have occurred due to cell-culture passage, as such features suggest the involvement of an immune system18.
Conclusions
In the midst of the global COVID-19 public-health emergency, it is reasonable to wonder why the origins of the pandemic matter. Detailed understanding of how an animal virus jumped species boundaries to infect humans so productively will help in the prevention of future zoonotic events. For example, if SARS-CoV-2 pre-adapted in another animal species, then there is the risk of future re-emergence events. In contrast, if the adaptive process occurred in humans, then even if repeated zoonotic transfers occur, they are unlikely to take off without the same series of mutations. In addition, identifying the closest viral relatives of SARS-CoV-2 circulating in animals will greatly assist studies of viral function. Indeed, the availability of the RaTG13 bat sequence helped reveal key RBD mutations and the polybasic cleavage site.
The genomic features described here may explain in part the infectiousness and transmissibility of SARS-CoV-2 in humans. Although the evidence shows that SARS-CoV-2 is not a purposefully manipulated virus, it is currently impossible to prove or disprove the other theories of its origin described here. However, since we observed all notable SARS-CoV-2 features, including the optimized RBD and polybasic cleavage site, in related coronaviruses in nature, we do not believe that any type of laboratory-based scenario is plausible.
More scientific data could swing the balance of evidence to favor one hypothesis over another. Obtaining related viral sequences from animal sources would be the most definitive way of revealing viral origins. For example, a future observation of an intermediate or fully formed polybasic cleavage site in a SARS-CoV-2-like virus from animals would lend even further support to the natural-selection hypotheses. It would also be helpful to obtain more genetic and functional data about SARS-CoV-2, including animal studies. The identification of a potential intermediate host of SARS-CoV-2, as well as sequencing of the virus from very early cases, would similarly be highly informative. Irrespective of the exact mechanisms by which SARS-CoV-2 originated via natural selection, the ongoing surveillance of pneumonia in humans and other animals is clearly of utmost importance.
We thank all those who have contributed sequences to the GISAID database (https://www.gisaid.org/) and analyses to Virological.org (http://virological.org/). We thank M. Farzan for discussions, and the Wellcome Trust for support. K.G.A. is a Pew Biomedical Scholar and is supported by NIH grant U19AI135995. A.R. is supported by the Wellcome Trust (Collaborators Award 206298/Z/17/Z―ARTIC network) and the European Research Council (grant agreement no. 725422―ReservoirDOCS). E.C.H. is supported by an ARC Australian Laureate Fellowship (FL170100022). R.F.G. is supported by NIH grants U19AI135995, U54 HG007480 and U19AI142790.
Author information
Affiliations
Department of Immunology and Microbiology, The Scripps Research Institute, La Jolla, CA, USA
Kristian G. Andersen
Scripps Research Translational Institute, La Jolla, CA, USA
Kristian G. Andersen
Institute of Evolutionary Biology, University of Edinburgh, Edinburgh, UK
Andrew Rambaut
Center for Infection and Immunity, Mailman School of Public Health of Columbia University, New York, NY, USA
W. Ian Lipkin
Marie Bashir Institute for Infectious Diseases and Biosecurity, School of Life and Environmental Sciences and School of Medical Sciences, The University of Sydney, Sydney, Australia
Edward C. Holmes
Tulane University, School of Medicine, Department of Microbiology and Immunology, New Orleans, LA, USA
Robert F. Garry
Zalgen Labs, Germantown, MD, USA
Robert F. Garry
Corresponding author
Correspondence to Kristian G. Andersen.
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Competing interests
R.F.G. is co-founder of Zalgen Labs, a biotechnology company that develops countermeasures to emerging viruses.
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Σ.γ.: Με το πιο πάνω άρθρο όλα είναι τακτοποιημένα, οι κυβερνήσεις μπορεί να το χρησιμοποιούν για το καλό μας, οι γιατροί για να δικαιολογούν τον εμβολιασμό όλων μας για να “σωθούμε” και όσοι πεθαίνουν από τις παρενέργειες του “τσιμπήματος”, αυτό ¨οφείλεται σε άλλες αιτίες” πλην των εμβολίων. Δείτε την παρακάτω ανάρτηση:
Τρεις γιατροί που εργάζονταν στην Pfizer, ο Chris Crose, ο Nick Karl και ο Rahul Khandke απεκάλυψαν σε κρυφή κάμερα (ποιο νόημα έχει η κρυφή αφού ανέφεραν και τα ονόματά τους, σίγουρα ήσαν υπό απόλυση) τα αίσχη και τις παρανομίες της Pfizer όσον αφορά τα εμβόλια. Κάτι που δεν μας ξενίζει καθώς η ίδια Εταιρεία έχει πληρώσει εκατομμύρια δολάρια πρόστιμο για πολυάριθμες παραβιάσεις και παρανομίες που έχει κάνει.
57 Top Scientists And Doctors Release Shocking Study On COVID Vaccines
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A group of 57 leading scientists, doctors and policy experts has released a report calling in to question the safety and efficacy of the current COVID-19 vaccines and are now calling for an immediate end to all vaccine programs. We urge you to read and share this damning report.
There are two certainties regarding the global distribution of Covid-19 vaccines. The first is that governments and the vast majority of the mainstream media are pushing with all their might to get these experimental drugs into as many people as possible. The second is that those who are willing to face the scorn that comes with asking serious questions about vaccines are critical players in our ongoing effort to spread the truth.
You can read an advanced copy of this manuscript in preprint below. It has been prepared by nearly five dozen highly respected doctors, scientists, and public policy experts from across the globe to be urgently sent to world leaders as well as all who are associated with the production and distribution of the various Covid-19 vaccines in circulation today.
There are still far too many unanswered questions regarding the Covid-19 vaccines’ safety, efficacy, and necessity. This study is a bombshell that should be heard by everyone, regardless of their views on vaccines. There aren’t nearly enough citizens who are asking questions. Most people simply follow the orders of world governments, as if they have earned our complete trust. They haven’t done so. This manuscript is a step forward in terms of accountability and the free flow of information on this crucial subject. Please take the time to read it and share it widely.
SARS-CoV-2 mass vaccination: Urgent questions on vaccine safety that demand answers from international health agencies, regulatory authorities, governments and vaccine developers
Roxana Bruno1, Peter McCullough2, Teresa Forcades i Vila3, Alexandra Henrion-Caude4, Teresa García-Gasca5, Galina P. Zaitzeva6, Sally Priester7, María J. Martínez Albarracín8, Alejandro Sousa-Escandon9, Fernando López Mirones10, Bartomeu Payeras Cifre11, Almudena Zaragoza Velilla10, Leopoldo M. Borini1, Mario Mas1, Ramiro Salazar1, Edgardo Schinder1, Eduardo A Yahbes1, Marcela Witt1, Mariana Salmeron1, Patricia Fernández1, Miriam M. Marchesini1, Alberto J. Kajihara1, Marisol V. de la Riva1, Patricia J. Chimeno1, Paola A. Grellet1, Matelda Lisdero1, Pamela Mas1, Abelardo J. Gatica Baudo12, Elisabeth Retamoza12, Oscar Botta13, Chinda C. Brandolino13, Javier Sciuto14, Mario Cabrera Avivar14, Mauricio Castillo15, Patricio Villarroel15, Emilia P. Poblete Rojas15, Bárbara Aguayo15, Dan I. Macías Flores15, Jose V. Rossell16, Julio C. Sarmiento17, Victor Andrade-Sotomayor17, Wilfredo R. Stokes Baltazar18, Virna Cedeño Escobar19, Ulises Arrúa20, Atilio Farina del Río21, Tatiana Campos Esquivel22, Patricia Callisperis23, María Eugenia Barrientos24, Karina Acevedo-Whitehouse5,*
1Epidemiólogos Argentinos Metadisciplinarios. República Argentina. 2Baylor University Medical Center. Dallas, Texas, USA. 3Monestir de Sant Benet de Montserrat, Montserrat, Spain 4INSERM U781 Hôpital Necker-Enfants Malades, Université Paris Descartes-Sorbonne Cité, Institut Imagine, Paris, France. 5School of Natural Sciences. Autonomous University of Querétaro, Querétaro, Mexico. 6Retired Professor of Medical Immunology. Universidad de Guadalajara, Jalisco, Mexico. 7Médicos por la Verdad Puerto Rico. Ashford Medical Center. San Juan, Puerto Rico. 8Retired Professor of Clinical Diagnostic Processes. University of Murcia, Murcia, Spain 9Urologist Hospital Comarcal de Monforte, University of Santiago de Compostela, Spain. 10Biólogos por la Verdad, Spain. 11Retired Biologist. University of Barcelona. Specialized in Microbiology. Barcelona, Spain. 12Center for Integrative Medicine MICAEL (Medicina Integrativa Centro Antroposófico Educando en Libertad). Mendoza, República Argentina. 13Médicos por la Verdad Argentina. República Argentina. ´ 14Médicos por la Verdad Uruguay. República Oriental del Uruguay. 15Médicos por la Libertad Chile. República de Chile. 16Physician, orthopedic specialist. República de Chile. 17Médicos por la Verdad Perú. República del Perú. 18Médicos por la Verdad Guatemala. República de Guatemala. 19Concepto Azul S.A. Ecuador. 20Médicos por la Verdad Brasil. Brasil. 21Médicos por la Verdad Paraguay. 22Médicos por la Costa Rica. 23Médicos por la Verdad Bolivia. 24Médicos por la Verdad El Salvador.
* Correspondence: Karina Acevedo-Whitehouse, karina.acevedo.whitehouse@uaq.mx
Abstract
Since the start of the COVID-19 outbreak, the race for testing new platforms designed to confer immunity against SARS-CoV-2, has been rampant and unprecedented, leading to emergency authorization of various vaccines. Despite progress on early multidrug therapy for COVID-19 patients, the current mandate is to immunize the world population as quickly as possible. The lack of thorough testing in animals prior to clinical trials, and authorization based on safety data generated during trials that lasted less than 3.5 months, raise questions regarding the safety of these vaccines. The recently identified role of SARS-CoV-2 glycoprotein Spike for inducing endothelial damage characteristic of COVID-19, even in absence of infection, is extremely relevant given that most of the authorized vaccines induce the production of Spike glycoprotein in the recipients. Given the high rate of occurrence of adverse effects, and the wide range of types of adverse effects that have been reported to date, as well as the potential for vaccine-driven disease enhancement, Th2-immunopathology, autoimmunity, and immune evasion, there is a need for a better understanding of the benefits and risks of mass vaccination, particularly in the groups that were excluded in the clinical trials. Despite calls for caution, the risks of SARS-CoV-2 vaccination have been minimized or ignored by health organizations and government authorities. We appeal to the need for a pluralistic dialogue in the context of health policies, emphasizing critical questions that require urgent answers if we wish to avoid a global erosion of public confidence in science and public health.
Introduction
Since COVID-19 was declared a pandemic in March 2020, over 150 million cases and 3 million deaths have been reported worldwide. Despite progress on early ambulatory, multidrug-therapy for high-risk patients, resulting in 85% reductions in COVID-19 hospitalization and death [1], the current paradigm for control is mass-vaccination. While we recognize the effort involved in development, production and emergency authorization of SARS-CoV-2 vaccines, we are concerned that risks have been minimized or ignored by health organizations and government authorities, despite calls for caution [2-8].
Vaccines for other coronaviruses have never been approved for humans, and data generated in the development of coronavirus vaccines designed to elicit neutralizing antibodies show that they may worsen COVID-19 disease via antibody-dependent enhancement (ADE) and Th2 immunopathology, regardless of the vaccine platform and delivery method [9-11]. Vaccine-driven disease enhancement in animals vaccinated against SARS-CoV and MERS-CoV is known to occur following viral challenge, and has been attributed to immune complexes and Fc-mediated viral capture by macrophages, which augment T-cell activation and inflammation [11-13].
In March 2020, vaccine immunologists and coronavirus experts assessed SARS-CoV-2 vaccine risks based on SARS-CoV-vaccine trials in animal models. The expert group concluded that ADE and immunopathology were a real concern, but stated that their risk was insufficient to delay clinical trials, although continued monitoring would be necessary [14]. While there is no clear evidence of the occurrence of ADE and vaccine-related immunopathology in volunteers immunized with SARS-CoV-2 vaccines [15], safety trials to date have not specifically addressed these serious adverse effects (SAE). Given that the follow-up of volunteers did not exceed 2-3.5 months after the second dose [16-19], it is unlikely such SAE would have been observed. Despite92 errors in reporting, it cannot be ignored that even accounting for the number of vaccines administered, according to the US Vaccine Adverse Effect Reporting System (VAERS), the number of deaths per million vaccine doses administered has increased more than 10-fold. We believe there is an urgent need for open scientific dialogue on vaccine safety in the context of large-scale immunization. In this paper, we describe some of the risks of mass vaccination in the context of phase 3 trial exclusion criteria and discuss the SAE reported in national and regional adverse effect registration systems. We highlight unanswered questions and draw attention to the need for a more cautious approach to mass vaccination.
SARS-CoV-2 phase 3 trial exclusion criteria
With few exceptions, SARS-CoV-2 vaccine trials excluded the elderly [16-19], making it impossible to identify the occurrence of post-vaccination eosinophilia and enhanced inflammation in elderly people. Studies of SARS-CoV vaccines showed that immunized elderly mice were at particularly high risk of life-threatening Th2 immunopathology [9,20]. Despite this evidence and the extremely limited data on safety and efficacy of SARS-CoV-2 vaccines in the elderly, mass-vaccination campaigns have focused on this age group from the start. Most trials also excluded pregnant and lactating volunteers, as well as those with chronic and serious conditions such as tuberculosis, hepatitis C, autoimmunity, coagulopathies, cancer, and immune suppression [16-29], although these recipients are now being offered the vaccine under the premise of safety.
Another criterion for exclusion from nearly all trials was prior exposure to SARS-CoV-2. This is unfortunate as it denied the opportunity of obtaining extremely relevant information concerning post-vaccination ADE in people that already have anti-SARS-Cov-2 antibodies. To the best of our knowledge, ADE is not being monitored systematically for any age or medical condition group currently being administered the vaccine. Moreover, despite a substantial proportion of the population already having antibodies [21], tests to determine SARS-CoV-2-antibody status prior to administration of the vaccine are not conducted routinely.
Will serious adverse effects from the SARS-CoV-2 vaccines go unnoticed?
COVID-19 encompasses a wide clinical spectrum, ranging from very mild to severe pulmonary pathology and fatal multi-organ disease with inflammatory, cardiovascular, and blood coagulation dysregulation [22-24]. In this sense, cases of vaccine-related ADE or immunopathology would be clinically-indistinguishable from severe COVID-19 [25]. Furthermore, even in the absence of SARS-CoV-2 virus, Spike glycoprotein alone causes endothelial damage and hypertension in vitro and in vivo in Syrian hamsters by down-regulating angiotensin-converting enzyme 2 (ACE2) and impairing mitochondrial function [26]. Although these findings need to be confirmed in humans, the implications of this finding are staggering, as all vaccines authorized for emergency use are based on the delivery or induction of Spike glycoprotein synthesis. In the case of mRNA vaccines and adenovirus-vectorized vaccines, not a single study has examined the duration of Spike production in humans following vaccination. Under the cautionary principle, it is parsimonious to consider vaccine-induced Spike synthesis could cause clinical signs of severe COVID-19, and erroneously be counted as new cases of SARS-CoV-2 infections. If so, the true adverse effects of the current global vaccination strategy may never be recognized unless studies specifically examine this question. There is already non-causal evidence of temporary or sustained increases138 in COVID-19 deaths following vaccination in some countries (Fig. 1) and in light of Spike’s pathogenicity, these deaths must be studied in depth to determine whether they are related to vaccination.
Unanticipated adverse reactions to SARS-CoV-2 vaccines
Another critical issue to consider given the global scale of SARS-CoV-2 vaccination is autoimmunity. SARS-CoV-2 has numerous immunogenic proteins, and all but one of its immunogenic epitopes have similarities to human proteins [27]. These may act as a source of antigens, leading to autoimmunity [28]. While it is true that the same effects could be observed during natural infection with SARS-CoV-2, vaccination is intended for most of the world population, while it is estimated that only 10% of the world population has been infected by SARS-CoV-2, according to Dr. Michael Ryan, head of emergencies at the World Health Organization. We have been unable to find evidence that any of the currently authorized vaccines screened and excluded homologous immunogenic epitopes to avoid potential autoimmunity due to pathogenic priming.
Some adverse reactions, including blood-clotting disorders, have already been reported in healthy and young vaccinated people. These cases led to the suspension or cancellation of the use of adenoviral vectorized ChAdOx1-nCov-19 and Janssen vaccinesin some countries. It has now been proposed that vaccination with ChAdOx1-nCov-19 can result in immune thrombotic thrombocytopenia (VITT) mediated by platelet-activating antibodies against Platelet factor-4, which clinically mimics autoimmune heparin-induced thrombocytopenia [29]. Unfortunately, the risk was overlooked when authorizing these vaccines, although adenovirus-induced thrombocytopenia has been known for more than a decade, and has been a consistent event with adenoviral vectors [30]. The risk of VITT would presumably be higher in those already at risk of blood clots, including women who use oral contraceptives [31], making it imperative for clinicians to advise their patients accordingly.
At the population level, there could also be vaccine-related impacts. SARS-CoV-2 is a fast-evolving RNA virus that has so far produced more than 40,000 variants [32,33] some of which affect the antigenic domain of Spike glycoprotein [34,35]. Given the high mutation rates, vaccine-induced synthesis of high levels of anti-SARS-CoV-2-Spike antibodies could theoretically lead to suboptimal responses against subsequent infections by other variants in vaccinated individuals [36], a phenomenon known as “original antigenic sin” [37] or antigenic priming [38]. It is unknown to what extent mutations that affect SARS-CoV-2 antigenicity will become fixed during viral evolution [39], but vaccines could plausibly act as selective forces driving variants with higher infectivity or transmissibility. Considering the high similarity between known SARS-CoV-2 variants, this scenario is unlikely [32,34] but if future variants were to differ more in key epitopes, the global vaccination strategy might have helped shape an even more dangerous virus. This risk has recently been brought to the attention of the WHO as an open letter [40].
Discussion
The risks outlined here are a major obstacle to continuing global SARS-CoV-2 vaccination. Evidence on the safety of all SARS-CoV-2 vaccines is needed before exposing more people to the184 risk of these experiments, since releasing a candidate vaccine without time to fully understand the resulting impact on health could lead to an exacerbation of the current global crisis [41]. Risk-stratification of vaccine recipients is essential. According to the UK government, people below 60 years of age have an extremely low risk of dying from COVID-191 187 . However, according to Eudravigillance, most of the serious adverse effects following SARS-CoV-2 vaccination occur in people aged 18-64. Of particular concern is the planned vaccination schedule for children aged 6 years and older in the United States and the UK. Dr. Anthony Fauci recently anticipated that teenagers across the country will be vaccinated in the autumn and younger children in early 2022, and the UK is awaiting trial results to commence vaccination of 11 million children under 18. There is a lack of scientific justification for subjecting healthy children to experimental vaccines, given that the Centers for Disease Control and Prevention estimates that they have a 99.997% survival rate if infected with SARS-CoV-2. Not only is COVID-19 irrelevant as a threat to this age group, but there is no reliable evidence to support vaccine efficacy or effectiveness in this population or to rule out harmful side effects of these experimental vaccines. In this sense, when physicians advise patients on the elective administration of COVID-19 vaccination, there is a great need to better understand the benefits and risk of administration, particularly in understudied groups.
In conclusion, in the context of the rushed emergency-use-authorization of SARS-CoV-2 vaccines, and the current gaps in our understanding of their safety, the following questions must be raised:
Is it known whether cross-reactive antibodies from previous coronavirus infections or vaccine206 induced antibodies may influence the risk of unintended pathogenesis following vaccination with COVID-19?
Has the specific risk of ADE, immunopathology, autoimmunity, and serious adverse reactions been clearly disclosed to vaccine recipients to meet the medical ethics standard of patient understanding for informed consent? If not, what are the reasons, and how could it be implemented?
What is the rationale for administering the vaccine to every individual when the risk of dying from COVID-19 is not equal across age groups and clinical conditions and when the phase 3 trials excluded the elderly, children and frequent specific conditions?
What are the legal rights of patients if they are harmed by a SARS-CoV-2 vaccine? Who will cover the costs of medical treatment? If claims were to be settled with public money, has the public been made aware that the vaccine manufacturers have been granted immunity, and their responsibility to compensate those harmed by the vaccine has been transferred to the tax-payers?
In the context of these concerns, we propose halting mass-vaccination and opening an urgent pluralistic, critical, and scientifically-based dialogue on SARS-CoV-2 vaccination among scientists, medical doctors, international health agencies, regulatory authorities, governments, and vaccine developers. This is the only way to bridge the current gap between scientific evidence and public health policy regarding the SARS-CoV-2 vaccines. We are convinced that humanity deserves a deeper understanding of the risks than what is currently touted as the official position. An open scientific dialogue is urgent and indispensable to avoid erosion of public confidence in science and public health and to ensure that the WHO and national health authorities protect the interests of humanity during the current pandemic. Returning public health policy to evidence-based medicine, relying on a careful evaluation of the relevant scientific research, is urgent. It is imperative to follow the science.
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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Weisblum Y, Schmidt F, Zhang F, et al. Escape from neutralizing antibodies by SARS354 CoV-2 spike protein variants. Elife (2020) 9:e61312. doi:10.7554/eLife.61312
Vanden Bossche G (March 6, 2021) https://dryburgh.com/wp-356content/uploads/2021/03/Geert_Vanden_Bossche_Open_Letter_WHO_March_6_2021.pdf
Coish JM, MacNeil AJ. Out of the frying pan and into the fire? Due diligence warranted for ADE in COVID-19. Microbes Infect (2020) 22(9):405-406. doi:10.1016/j.micinf.2020.06.006
Figure legends
Figure 1. Number of new COVID-19 deaths in relation to number of people that have received at least one vaccine dose for selected countries. Graph shows data from the start of vaccination to May 3rd 365 , 2021. A) India (9.25% of population vaccinated), B) Thailand (1.58% of population vaccinated), C) Colombia (6.79% of population vaccinated), D) Mongolia (31.65% of population vaccinated), E) Israel (62.47% of population vaccinated), F) Entire world (7.81% of population vaccinated). Graphs were built using data from Our World in Data (accessed 4 May 2021) https://github.com/owid/covid-19-data/tree/master/public/data/vaccinations
COVID Vaccine Biggest Biological Catastrophe With Medicinal Product In Human History – Dr. McCullough
Red Voice Media said infront of
What happened? Well, we now know that this early safety warning in this peer-review publication from Jessica Rose clearly failed. Look how high those mortality numbers were by April. It clearly failed. We had Americans dying after vaccination. It was obvious. This is an obvious data signal. This is obvious. All experts agree it’s obvious. Now, as of September 24th, it’s raced up to 15,937 Americans have died. Over 250,000 Americans, after the vaccine, have been hospitalized, gone to the urgent care or office visits. You can see the temporal relationship on the bottom bar graph.
Τι συνέβη? Λοιπόν, γνωρίζουμε τώρα ότι αυτή η έγκαιρη προειδοποίηση ασφάλειας σε αυτήν τη δημοσίευση αξιολόγησης από ομοτίμους συναδέλφους της Jessica Rose απέτυχε σαφώς. Κοιτάξτε πόσο υψηλοί ήταν οι αριθμοί θνησιμότητας μέχρι τον Απρίλιο. Σαφώς απέτυχε. Είχαμε Αμερικανούς να πεθαίνουν μετά τον εμβολιασμό. Αυτό ήταν προφανές. Αυτό είναι ένα προφανές προειδοποιητικό σήμα δεδομένων. Όλοι οι ειδικοί συμφωνούν ότι είναι προφανές. Τώρα, από τις 24 Σεπτεμβρίου, έχουν ανέλθει σε συχνότητα θανάτων έως και 15.937 Αμερικανοί έχουν πεθάνει. Πάνω από 250.000 Αμερικανοί, μετά το εμβόλιο, νοσηλεύτηκαν, πήγαν σε επείγουσα περίθαλψη ή επισκέψεις στο γραφείο. Μπορείτε να δείτε τη χρονική σχέση στο γράφημα εδώ κάτω
Y.ou can see that sharp spike upwards. Sadly, we have over 20,000 Americans that the CDC tells us are permanently disabled after the vaccine. That’s bigger than some major Cancer groups. That’s bigger than some major Cancer groups. The disability that we are going to see due to these vaccines will go down in history as an unbelievable atrocity. I made a presentation to The Heritage Foundation in Washington that provides a lot of oversight to the House and the Senate, as well as the agencies, and I made this presentation.
Μπορείτε να δείτε αυτήν την απότομη κορυφή προς τα πάνω. Δυστυχώς, έχουμε πάνω από 20.000 Αμερικανούς για τους οποίους το CDC μας λέει ότι είναι μόνιμα ανάπηροι μετά το εμβόλιο. Αυτό το ποσοστό είναι μεγαλύτερο από μερικές μεγάλες ομάδες καρκίνου. Η αναπηρία που πρόκειται να δούμε λόγω αυτών των εμβολίων θα μείνει στην ιστορία ως μια απίστευτη θηριωδία. Έκανα μια παρουσίαση στο The Heritage Foundation στην Ουάσινγκτον που παρέχει μεγάλη ενημέρωση στο Σώμα των Αντιπροσώπων και στη Γερουσία, καθώς και τους σχετικούς οργανισμούς, και έκανα αυτήν την παρουσίαση.
You could hear a pin drop when I was done, pin drop. And finally, one of the former presidents of the American Medical Association said, Dr. McCullough, we have the biggest biological catastrophe on our hands in human history with a medicinal product, and we’ve had two administrations buy into it. We’ve had all the houses of legislation buy into it. We have the entire medical established buy into it and the whole media and no one knows how to stop it. No one knows how to stop this freight train, and we’re all witnessing it right now.” –
Θα μπορούσατε να ακούσετε ως και μια πτώση καρφίτσας από την απόλυτη ησυχία που επικρτούσε όταν τελείωσα. Και τέλος, ένας από τους πρώην προέδρους της Αμερικανικής Ιατρικής Εταιρείας είπε: Δρ ΜακΚάλοου, έχουμε τη μεγαλύτερη βιολογική καταστροφή στην ανθρώπινη ιστορία με ένα φαρμακευτικό προϊόν και είχαμε δύο Κυβερνήσεις του Τραμπ και του Μπάϊντεν να το αγοράσουν.
Είχαμε εξαγοράσει όλα τα νομοθετικά κέντρα. Έχουμε εξαγοράσει ολόκληρο το ιατρικό κατεστημένο και όλα τα μέσα ενημέρωσης και κανείς δεν ξέρει πώς να το σταματήσει. Κανείς δεν ξέρει πώς να σταματήσει αυτό το φορτηγό τρένο και όλοι το παρακολουθούμε αυτή τη στιγμή να συνεχίζει το δρόμο προς την καταστροφή».
The science that can’t be questioned is PROPAGANDA! Επιστήμη που δεν θέτει ερωτήματα είναι προπαγάνδα.
The real science and data prove that something’s not right with the official stories shared by the corrupted MSM. Η πραγματική επιστήμη και τα ορθά δεδομένα αποδεικνύουν ότι κάτι δεν είναι σωστό και οι επίσημες ιστορίες (πληροφορίες) διαδίδονται από διεφθαρμένα MSM.
They constantly share the phrases like ‘’Pandemic of the Unvaccinated,’’ ‘’Follow The Science,’’ ‘’Trust The Science.’’ Αυτοί συνεχώς χρησιμοποιούν φράσεις όπως “Η Πανδημία οφείλεται στους ανεμβολίαστους”,
“Ακολουθείστε τι λέει η επιστημη”, “εμπιστευτείτε την επιστήμη”.
However, they very rarely present us the growing number of scientists, experts, doctors, healthcare workers’ information about the pandemic! Why? I guess it is because their statements don’t fit the officials’ narration.Εν τούτοις πολύ σπάνια μας παρουσιάζουν τον ολοένα αυξανόμενο αριθμό επιστημόνων, ειδικών, ιατρών, νοσηλευτών, που πληροφορούν περί της πανδημίας! Γιατί; Πιστεύω διότι οι δηλώσεις που κάνουν δεν συμφωνούν με τους πολιτικούς και την επίσημη άποψη.
If the MSM shares the actual truth, Big Pharma will lose billions! Αν τα Μέσα Μαζικής Επικοινωνίας μεταδώσουν την πραγματική αλήθεια η Big Pharma θα χάσει δισεκατομμύρια κέρδη..
Dr. Peter McCullough is but one of those Big Pharma people collaborating with Big Media and Big Tech trying to silence and censor. Ο ιατρός Peter McCullough είναιν ένας από εκείνους που οι 4 μεγάλες φαρμακευτικές Εταιρείες και όσοι συνεργάζονται με αυτές με τα μεγάλα Μέσα Ενημέρωσης και τις μεγάλες τεχνικές υπηρεσίες κάνουν τα αδύνατα δυνατά για να τους φιμώσουν και λογοκρίνουν.
Δείτε το βίντεο πιο κάτω την μετάφραση την διαβάσατε ήδη πιο πάνω:
What happened? Well, we now know that this early safety warning in this peer-review publication from Jessica Rose clearly failed. Look how high those mortality numbers were by April. It clearly failed. We had Americans dying after vaccination. It was obvious. This is an obvious data signal. This is obvious. All experts agree it’s obvious. Now, as of September 24th, it’s raced up to 15,937 Americans have died. Over 250,000 Americans, after the vaccine, have been hospitalized, gone to the urgent care or office visits. You can see the temporal relationship on the bottom bar graph. You can see that sharp spike upwards. Sadly, we have over 20,000 Americans that the CDC tells us are permanently disabled after the vaccine. That’s bigger than some major Cancer groups. That’s bigger than some major Cancer groups. The disability that we are going to see due to these vaccines will go down in history as an unbelievable atrocity. I made a presentation to The Heritage Foundation in Washington that provides a lot of oversight to the House and the Senate, as well as the agencies, and I made this presentation. You could hear a pin drop when I was done, pin drop. And finally, one of the former presidents of the American Medical Association said, Dr. McCullough, we have the biggest biological catastrophe on our hands in human history with a medicinal product, and we’ve had two administrations buy into it. We’ve had all the houses of legislation buy into it. We have the entire medical established buy into it and the whole media and no one knows how to stop it. No one knows how to stop this freight train, and we’re all witnessing it right now.” – Dr. Peter McCullough
I think every person has the reasonability to amplify the credible voices that have been silenced and stop them from informing you about the absolute truth!
Dr. McCullough is one of those persons.
Rumble — Ο Δρ McCullough έχει δημοσιεύσει ευρέως σε διάφορα ιατρικά θέματα με πάνω από 1000 δημοσιεύσεις και πάνω από 600 αναφορές στην Εθνική Βιβλιοθήκη Ιατρικής. Στις εργασίες του περιλαμβάνεται το κεφάλαιο “Σύνδεση μεταξύ νεφροπάθειας και καρδιοπάθειας” στο Εγχειρίδιο Καρδιοπάθειας του Braunwald. Ο Δρ McCullough είναι ιδρυτής και σημερινός πρόεδρος της Αμερικανικής Καρδιονεφρικής Εταιρείας, μιας οργάνωσης που έχει ως στόχο να φέρει σε επαφή καρδιολόγους και νεφρολόγους για να εργαστούν πάνω στο αναδυόμενο πρόβλημα των καρδιονεφρικών συνδρόμων. Έργα του έχουν δημοσιευθεί στο New England Journal of Medicine, στο Περιοδικό της Αμερικανικής Ιατρικής Ένωσης, στο Lancet, στο British Medical Journal και σε άλλα κορυφαία περιοδικά παγκοσμίως. Είναι αρχισυντάκτης του περιοδικού Ανασκοπήσεις στην Καρδιαγγειακή Ιατρική (Reviews in Cardiovascular Medicine) και ανώτερος συνεργάτης του Αμερικανικού Περιοδικού Καρδιολογίας. Είναι μέλος των συντακτικών επιτροπών πολλών ιατρικών περιοδικών. Ο Dr. McCullough έχει κάνει παρουσιάσεις σχετικά με την βελτίωση της ιατρικής σε όλο τον κόσμο και έχει υπάρξει προσκεκλημένος ομιλητής στην Ακαδημία Επιστημών της Νέας Υόρκης, στα Εθνικά Ινστιτούτα Υγείας, στον Οργανισμό Τροφίμων και Φαρμάκων των ΗΠΑ (FDA) και στον Ευρωπαϊκό Οργανισμό Φαρμάκων. Έχει διατελέσει μέλος ή πρόεδρος των επιτροπών παρακολούθησης της ασφάλειας δεδομένων σε 24 τυχαιοποιημένες κλινικές έρευνες.
(πηγή βιογραφικού:
Αυτό το βίντεο που σας μεταφράσαμε verbatum είναι από πρόσφατες δηλώσεις του για τα εμβόλια του κορωνοϊού (12 Οκτωβρίου 2021).
Σχετικά δημοσιεύματα στα αγγλικά:
α) Dr. McCullough With Breaking Announcement: C-19 Vaccine- The Biggest Biological Catastrophe!
β) 57 Top Scientists and Doctors: Stop All Covid Vaccinations
Dr.P.McCullough: «Παίξαμε με την μητέρα φύση – Οι κυβερνήσεις πίεσαν για μαζικό εμβολιασμό και “αχρήστεψαν” τα εμβόλια»
«Μας είχαν προειδοποιήσει: Μην εμβολιάζεστε εν μέσω πανδημίας!»
Ο Dr. Peter A McCullough σύμβουλος Καρδιολόγος και Προϊστάμενος του Τμήματος Διατροφής και Προληπτικής Ιατρικής στο Νοσοκομείο William Beaumont στο Royal Oak, του Μίσιγκαν στις ΗΠΑ υποστήριξε ότι με τον μαζικό εμβολιασμό οι κυβερνήσεις «αχρήστευσαν» τα εμβόλια, καθώς ο ιός βρήκε τρόπο να ξεφύγει.
Ο γιατρός αναφέρθηκε σε συνέντευξη του στις δηλώσεις που είχαν κάνει επιστήμονες τους προηγούμενους μήνες και έλεγαν ότι δεν πρέπει να γίνουν μαζικοί εμβολιασμοί εν μέσω πανδημίας.
Όπως τόνισε στις δηλώσεις του ο λόγος που δημιουργήθηκε η μετάλλαξη Δέλτα, αλλά και άλλες μεταλλάξεις που καθιστούν άχρηστα τα υπάρχοντα εμβόλια είναι ακριβώς αυτός.
Ο γιατρός έδειξε τα στοιχεία του CDC για τα κρούσματα και θανάτους με Δέλτα σε εμβολιασμένους και μη και τόνισε τα εξής:
«Με το να ωθούν για μαζικούς εμβολιασμούς, οι κυβερνήσεις έχουν δημιουργήσει εξελικτικές πιέσεις στον SARS-coV-2. Και κάποιοι μάς προειδοποίησαν γι’ αυτό….
Ο Geert Vanden Bossche, ο Michael Yeadon, ο Sucharit Bhakdi, ο dr Luc Montagnier μας προειδοποίησαν γι’ αυτό. Μην εμβολιάζετε εν μέσω πανδημίας!
Είπαν ότι ο ιός θα ανακαλύψει αυτά τα εμβόλια και θα βρει τρόπο (να ξεφύγει). Η παραλλαγή Δέλτα είναι ακριβώς αυτό…. Έχει επιτύχει αντιγονική διαφυγή.
Παίξαμε με την μητέρα φύση! Είμαστε σε ποσοστό 60% εμβολιασμένοι. Έτσι τώρα η Δέλτα ήρθε για να μείνει μέχρι να αλλάξουν τα εμβόλια….
Πρόσφατη εργασία, που μόλις κυκλοφόρησε απ’ το Πανεπιστήμιο Davis της Καλιφόρνια, δείχνει ότι σε εμβολιασμένους και ανεμβολίαστους τα ιικά φορτία είναι υψηλά και ίδια στις δύο ομάδες.
Είναι ξεκάθαρο ότι τα εμβόλια δεν κάνουν απολύτως τίποτα για να βοηθήσουν να μειωθεί η φορεία του ιού!
Ένα εμβολιασμένο άτομο απειλεί εξίσου κάποιον που δεν έχει εμβολιαστεί και δεν έχει νοσήσει. Αυτό που μας έδειξαν ο Niesen και οι συνεργάτες του απ’ την κλινική Mayo είναι ότι μόλις ξεκινήσαμε να εμβολιάζουμε και φτάσαμε στο 25% εμβολιασμένων, η ποικιλομορφία άρχισε να πέφτει, ότι ο αριθμός των διαφορετικών στελεχών που κατηγοριοποιούσε το CDC κάθε 2 βδομάδες άρχισε να κατακρημνίζεται, επειδή αρχίσαμε να παίζουμε με την μητέρα φύση και δεν θα ‘πρεπε να το κάνουμε αυτό!!!!».
Colorized scanning electron micrograph of Marburg virus particles (blue) both budding and attached to the surface of infected VERO E6 cells (orange). Image captured and color-enhanced at the NIAID Integrated Research Facility in Fort Detrick, Maryland. Credit: NIAID.
A deadly cousin of Ebola, Marburg can kill nine out of ten people it infects, and international travel has taken it from Africa to Europe twice in the past 40 years. Will increasing globalisation make this virus more likely to erupt around the world?
In August 1967, a cluster of patients in Marburg and Frankfurt, in Germany, and in Belgrade (then Yugoslavia, now Serbia), began showing symptoms of an infectious disease – a high fever, chills, muscle ache, and vomiting. The patients worsened over the next few days, until they began bleeding from every orifice in their body, including needle puncture wounds. In total 31 people died.
Three months after this outbreak, virologists in Marburg had discovered the first filovirus, a cousin of the equally-deadly Ebola virus. The virus had been carried by infected African green monkeys from Uganda.
Avoiding handling or eating bush meat is also critical to avoid any potential infection that could spread from animals.
After this first sighting, the virus was then mostly seen in African countries, in bat-infested caves or mines. About 40 years later, however, the virus re-emerged in Europe through a traveler returning to the Netherlands from a trip to Uganda where she had been visiting caves.
The largest known outbreak of Marburg virus, in Angola in 2004, infected over 250 people and had a 90 percent fatality rate.
Marburg virus can persist in the eyes and testes of people who have recovered, and in pregnant women it can persist in the placenta and amniotic fluid as well as breast milk. This can be extremely dangerous. In early 2021, there were reports that Ebola, closely related to Marburg, could lay dormant in people only to emerge many months after an epidemic had ended, triggering another outbreak.
Disease: Marburg
Where is it circulating? Most outbreaks have been in Africa, with cases reported in Angola, the Democratic Republic of the Congo, Kenya, South Africa, Uganda and Zimbabwe. However, there have been outbreaks in Europe and the USA.
Pandemic threat: As Marburg virus can spread from human to human through contact of bodily fluids, much like Ebola. As outbreaks in Europe and the US have already shown, increasing globalisation and international travel mean that the risk for global spread is high, especially when the incubation period could be up to three weeks. This could be disastrous given its high death rate.
How is it spread? The Egyptian rousette fruit bats often harbour the virus. African green monkeys have in the past spread the virus to people in Uganda, but pigs can also become infected and can be a source of infection. Marburg virus is spread through direct contact (through broken skin or mucous membranes) with the blood, secretions, organs or other bodily fluids of infected people, and also through any materials such as bedding, that have been contaminated with the infected fluids. As a result, health workers have often become infected by treating patients with Marburg virus. Burial ceremonies in which people have direct contact with the body can also drive the spread of the virus.
Case fatality rate: Marburg is one of the deadliest viruses we know of, killing as many as 88% people it infects.
Incubation period: The incubation varies from as short as two days to up to 21 days, though some studies have suggested the virus can incubate for as long as 26 days.
Symptoms: Marburg virus begins with a fever, severe headache and muscle pains. This is often followed by watery diarrhoea, stomach pain, nausea and vomiting, accompanied by extreme exhaustion and lethargy. Many people go on to develop severe viral haemorrhagic fever, and in severe cases have blood in their vomit and faeces, and may bleed from their nose, gums and vagina. The onslaught of the virus is so extreme that most people die 8-9 days after infection, often because of extreme loss of blood.
Diagnosis: Marburg can be difficult to distinguish clinically from other diseases, such as malaria, typhoid fever, meningitis and other viral haemorrhagic fevers. Diagnosis can be confirmed using techniques that detect the presence of immune response to the virus, such as antibody-capture enzyme-linked immunosorbent assay (ELISA), or the presence of virus in people presenting with symptoms, via antigen-capture detection tests, reverse transcriptase polymerase chain reaction (RT-PCR) assay, or virus isolation by cell culture. However, often none of these diagnostic tools are available in the countries with highest risk of Marburg outbreaks. In addition to having the diagnostic tests available, countries need to have laboratories that can ensure maximum biological containment conditions due to the fact that the samples are an extreme biohazard risk.
Are there vaccines or treatments, or ongoing R&D?
There are currently no specific therapeutics for Marburg virus. However, supportive care including rehydration with oral or intravenous fluids can improve survival. This can mean maintaining oxygen status and blood pressure, replacing lost blood and clotting factors, and treating any complicating infections. Potential treatments, including blood products, immune therapies and drug therapies, are currently being assessed. Marburg virus vaccine candidates are being investigated, and in 2019, for example, IAVI (the International AIDS Vaccine Initiative) began researching a recombinant vesicular stomatitis virus (VSV) vector Marburg virus vaccine candidate, called rVSVΔG-MARV-GP. Another vaccine candidate MVA-BN Filo containing both Marburg and Ebola virus antigens could potentially protect against both haemorrhagic viruses. It is currently in phase 3 trials, and seems to trigger good immunity against the Ebola Zaire strain but it has not yet been tested against Marburg virus.
How could we lower the risk of it becoming a pandemic?
Since Marburg virus can spread between people, extremely stringent infection control measures are needed to avoid people being in any contact with each other, to ensure any laboratory samples are disposed of carefully, and to ensure safe burial procedures. Avoiding handling or eating bush meat is also critical to avoid any potential infection that could spread from animals. International travel is a major risk factor for the spread of Marburg virus beyond Africa and rapid diagnostics to ensure that cases are picked up before people carry the virus to other countries will be important.
Κυρίες και Κύριοι. Θα ήθελα να προετοιμαστείτε συναισθηματικά και πνευματικά για ό,τι πρόκειται να ξεδιπλωθεί εδώ στη Γη. Είναι έτοιμοι να μας θυσιάσουν στον Μολώχ. Πιστεύετε τώρα που είδαν τι επιτυχία είχε ο COVID-19 θα σταματήσει το Παγκόσμιο Διευθυντήριο να εξαπολύει τα βιολογικά του όπλα;
Η επόμενη Πανδημία πρόκειται να είναι ο ιός Marburg. Πρόκειται για αιμορραγικό πυρετό παρόμοιο με τον Έμπολα με ποσοστό θνησιμότητας 88%. Τώρα καταρχάς μην ανησυχείτε, όσοι προωθούν αυτήν την ατζέντα δεν θα κυκλοφορήσουν τίποτα που θα τους σκότωνε. Αυτό θα είναι ψεύτικο Marburg. Πολλοί από τους τραυματίες του εμβολίου covid εμφανίζουν θρόμβους και ανεξέλεγκτη αιμορραγία. Αυτό θα ισχυριστεί ότι είναι Marburg. Το GAVI και το WEF κάνουν ήδη ανακοινώσεις σχετικά με τον ιό. Έχουν ήδη αναπτύξει ένα τεστ PCR για το Marburg, παρόλο που δεν υπάρχει ακόμη επίσημη «πανδημία». Το πιο ανησυχητικό είναι ότι ήδη σπεύδουν να βρουν ένα «εμβόλιο» για τον Marburg. Ακόμα πιο ανησυχητικό, το κύριο συστατικό του νέου εμβολίου είναι η Ρικίνη. Ένα από τα πιο τοξικά δηλητήρια στον πλανήτη. Θα επιτρέψουν στους εμβολιασμένους να ταξιδέψουν παγκοσμίως αυτά τα Χριστούγεννα. Θα χρειαστούν ένα κάλυμμα για άτομα που φέρνουν τον «ιό» πίσω στις χώρες τους. Μια νέα πανδημία θα κηρυχθεί, θα ισχυριστούν ότι υπάρχει ασυμπτωματική εξάπλωση. Τα μέσα ενημέρωσης θα εκτοξεύσουν τον φόβο πιο σκληρά από ό,τι έχουμε δει ποτέ. Οι άνθρωποι θα χάσουν το μυαλό τους πιστεύοντας ότι υπάρχει πανδημία για κάτι με πιθανότητα 88% να τους κάνει να αιμορραγήσουν και να πεθάνουν. Σε αυτό το στάδιο τα τρυπήματα θα είναι υποχρεωτικά και οι αστυνομικές και στρατιωτικές ομάδες θα είναι σε πλήρη ισχύ προσπαθώντας να «σώσουν την ανθρωπότητα» και να παρασύρουν ανθρώπους που αρνούνται το εμβόλιο του Μάρμπουργκ στα χτισμένα τώρα στρατόπεδα συγκέντρωσης για να τους κάνουν ενέσεις. Το «εμβόλιο» της Ρικίνης είναι ο πραγματικός θάνατος και θα υπάρχουν δισεκατομμύρια άνθρωποι που θα το κάνουν και θα αγωνίζονται για να βρεθούν στην πρώτη γραμμή. Αν νομίζετε ότι έχετε δει πανικό κατά τη διάρκεια του COVID, πολλαπλασιάστε το με 20 ή 30 Χ με αυτό που σχεδιάζεται και έρχεται. Κρατήστε τις οικογένειές σας κοντά. https://threadreaderapp.com/thread/1444688482409107456.html
Μην ταράζεστε, άλλοι φροντίζουν για τον Νέο Θαυμαστό Κόσμο που θα ζήσετε και σεις και τα παιδιά σας από δω και στο εξής. Και όπως λέει ο πατέρας του χαζοβιβλίου “Great Reset” Klaus Swabb ποτέ δεν θα επιστρέψουμε στην παλιά κανονικότητα.
Μια νέα αρχή ξεκινάει για την ανθρωπότητα, ένας καθαρά σιωνιστικός μεσσιανικός τρόπος του ζειν. Όπως το έχει προβλέψει ο Ιωάννης στην “Αποκάλυψή” του και πολλοί άλλοι θεοφώτιστοι πατέρες της εκκλησίας μας όπως ο Άγιος Ανδρέας Καισαρείας, ο Άγιος Ιππόλυτος, ο Όσιος Εφραιμ ο Σύρος, Άγιος Μεθόδιος Πατάρων, κι ο Άγιος Αρέθας εκ Καισαρείας, ο Όσιος Νείλος και ο προφήτης Δανιήλ και άλλοι[1].
Τώρα μας προέκυψε και η κυρία Γιάννα Αγγελοπούλου η οποία μυημένη στις αρχές της Νέας Τάξης Πραγμάτων πολύτιμη παγκοσμιοποιήτρια, είπε αλήθειες καθόλου τυχαίες οι οποίες βρίσκονται πολύ κοντά στην πραγματοποίησή τους. ‘Ακούστε τη και χαρείτε την…
https://www.youtube.com/watch?v=KAir7PKzgSo
Καταλάβατε; με ένα τσιπάκι, κάνοντας σκόνη τα προσωπικά μας δεδομένα, εμάς των πληβείων εννοεί, θα λυθούν (γι’ αυτούς, το Παγκόσμιο Διευθυντήριο) όλα τα προβλήματα.
Τι άλλο περιμένετε; Τώρα το ξέρετε που θα βαδίσουν τα πράγματα αν τους αφήσετε και κοιμάσθε ύπνον βαρύ.
Γεώργιος Ευαγγελάτος
[1]Ευαγγελάτου Γεωργίου: Η ΑΠΟΚΑΛΥΨΗ ΤΟΥ ΙΩΑΝΝΗ ΑΠΟΚΑΛΥΠΤΕΤΑΙ, Εκδόσεις Στερέωμα, Θεσσαλονίκη 2005.
O Γιώργος Ευαγγελάτος ψυχίατρος – ψυχαναλυτής παρέχει ψυχιατρική βοήθεια και συμβουλευτική σε ανθρώπους που αντιμετωπίζουν προσωπικές ή οικογενειακές ψυχολογικές και ψυχοκοινωνικές δυσκολίες. Μάθετε περισσότερα για εμάς εδώ.
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